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Addictive Substances and Neurological Disease

- Alcohol, Tobacco, Caffeine, and Drugs of Abuse in Everyday Lifestyles

Forfatter: info mangler
Bog
  • Format
  • Bog, hardback
  • Engelsk

Beskrivelse

Addictive Substances and Neurological Disease: Alcohol, Tobacco, Caffeine, and Drugs of Abuse in Everyday Lifestyles is a complete guide to the manifold effects of addictive substances on the brain, providing readers with the latest developing research on how these substances are implicated in neurological development and dysfunction. Cannabis, cocaine, and other illicit drugs can have substantial negative effects on the structure and functioning of the brain. However, other common habituating and addictive substances often used as part of an individual’s lifestyle, i.e., alcohol, tobacco, caffeine, painkillers can also compromise brain health and effect or accentuate neurological disease. This book provides broad coverage of the effects of addictive substances on the brain, beginning with an overview of how the substances lead to dysfunction before examining each substance in depth. It discusses the pathology of addiction, the structural damage resulting from abuse of various substances, and covers the neurobiological, neurodegenerative, behavioral, and cognitive implications of use across the lifespan, from prenatal exposure, to adolescence and old age. This book aids researchers seeking an understanding of the neurological changes that these substances induce, and is also extremely useful for those seeking potential treatments and therapies for individuals suffering from chronic abuse of these substances.

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Detaljer
  • SprogEngelsk
  • Sidetal414
  • Udgivelsesdato23-02-2017
  • ISBN139780128053737
  • Forlag Academic Press Inc
  • FormatHardback
Størrelse og vægt
  • Vægt1430 g
  • coffee cup img
    10 cm
    book img
    21,6 cm
    27,6 cm

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    Motivation Aggression Intervention Ethanol Drug-addiction Addiction Alcohol Alcoholism Reward Brain Rat Smoking Aging Cognition Gene expression. Inflammation Motor skills Neurophysiology Hallucinogens Neuroplasticity Serotonin Toxicity Binge drinking Metabolism Caffeine Homocysteine Hypothalamus Ketamine Auditory perception Neurons Motor Cortex Vitamin C Animal models Dopamine Nicotine Brain stimulation Pathophysiology Toxicology Sleep Myelination Magnetic resonance imaging Fetal alcohol syndrome Cerebellum Behavior Nicotine addiction Ghrelin Nitric oxide Blood cholesterol Central Nervous System Autophagy Hippocampus Histamine Trauma Vascular smooth muscle Astrocytes Deep Brain Stimulation Amygdala Energy drinks Executive Function Zebrafish Neuroimaging neonatal Habits Reactive Oxygen Species Human FASD NMDA Cognitive Distortion Oxidative damage Alcohol abuse Striatum Neuroinflammation Psychostimulants Visual stimuli Neurotoxicity Environmental Tobacco Smoke Reward Deficiency Syndrome Impulsivity Intoxication Brain structure Glial cells FAS Cortex Neuroimmune C-fos Brain development Brain disorders Neurodegeneration Insula Alcohol consumption Physical activity Novel psychoactive substances MiRNAs Outcome Brain Oscillations Prenatal Spinal cord injury Neurotransmitter Lateralization Goal-oriented FMRI Nicotine dependence Default Mode Network Craving Antisocial TLR4 Genetic association Inhibitory control Nucleus Accumbens Action schemata Alcohol-preferring (P) rats Antenatal alcohol exposure Automatized behavior Behavioral activation system Behavioral paradigms Basal forebrain Caffeinated alcohol Central nucleus of the amygdala Cellular and molecular disorders Central nervous system (CNS)Cerebellum Central pontine and extrapontine myelinolysis Cue reactivity Ethanol binding EtOH)Nervous tissue Executive control network Co-abuse GHS-R1a Ethanol brain atrophy GFAP histamine receptors �Cys-loop� Action knowledge Histaminergic neurons structure Histological changes Dissociative drugs KTSND Ethanol receptor Mesolimbic circuitry Middle temporal gyrus ligand-gated ion channels superfamily Morphological disturbances Maternal Smoking Medial prefrontal cortex musical notes Paradise Lost Theory Nicotinic cholinergic receptors Glutamate uptake Quantitative endurability Parietal Lobe Inferior Temporal Cortex Reward bias Serotonin-3 receptor SNCA Sedative substances Sensitization theory Salience Network Synaptic proteins Tool use skills Wernicke�Korsakoff syndrome Nicotine abuse nicotine exposure Monosubstance use Premotor cortex Subthalamic Nucleus Twin study Polysubstance use Self-reproductive reward Sensitivity to contrast Vitamin A�vitamin E Wernicke's encephalopathy Splice variants S100B and behavior Synthetic Cannabinoids Tryptophan hydroxylase 2 Ventral Tegmental Area
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